{"id":44217,"date":"2020-09-29T04:30:46","date_gmt":"2020-09-29T02:30:46","guid":{"rendered":"https:\/\/rina.naturopatiadigital.eu\/?p=44217"},"modified":"2020-10-13T18:46:49","modified_gmt":"2020-10-13T16:46:49","slug":"44217","status":"publish","type":"post","link":"https:\/\/rina.naturopatiadigital.eu\/?p=44217","title":{"rendered":"\u00c1rea de conocimiento: Tecnolog\u00eda Naturop\u00e1tica o Naturopat\u00eda Aplicada: M\u00f3dulo: Naturopat\u00eda Alimentaria &#8211; TROFOLOGIA: \u00abVitamina C y disfunci\u00f3n microvascular en la inflamaci\u00f3n sist\u00e9mica\u00bb"},"content":{"rendered":"<p><a href=\"https:\/\/1.bp.blogspot.com\/-CcMpo8nzSd0\/X3X03lu2UrI\/AAAAAAAAf4Y\/pkFScg5lNzU86c4hfjXq6JLy3tZv26I-QCLcBGAsYHQ\/s435\/Antioxidants.jpg\"><img decoding=\"async\" class=\"alignleft\" src=\"https:\/\/1.bp.blogspot.com\/-CcMpo8nzSd0\/X3X03lu2UrI\/AAAAAAAAf4Y\/pkFScg5lNzU86c4hfjXq6JLy3tZv26I-QCLcBGAsYHQ\/s320\/Antioxidants.jpg\" height=\"320\" border=\"0\" data-original-height=\"435\" data-original-width=\"303\" \/><\/a>Tyml, K. (2017).\u00a0<a href=\"https:\/\/www.mdpi.com\/2076-3921\/6\/3\/49\/htm\">Vitamin C and microvascular dysfunction in systemic inflammation<\/a>.\u00a0<i>Antioxidants<\/i>, 6(3), 49.<\/p>\n<p>La sepsis, disfunci\u00f3n org\u00e1nica potencialmente mortal causada por una respuesta disfuncional del hu\u00e9sped a la infecci\u00f3n, se asocia con una alta mortalidad. Una estrategia prometedora para mejorar el resultado es inyectar a los pacientes por v\u00eda intravenosa con ascorbato (vitamina C).<\/p>\n<p>En modelos animales de sepsis, esta inyecci\u00f3n mejora la supervivencia y, entre otros, la funci\u00f3n microvascular. Esta revisi\u00f3n examina nuestro trabajo reciente que aborda la capacidad del ascorbato para inhibir la disfunci\u00f3n arteriolar y el taponamiento capilar en la sepsis. La disfunci\u00f3n arteriolar incluye vasoconstricci\u00f3n \/ dilataci\u00f3n alterada (revisada anteriormente) y conducci\u00f3n alterada de vasoconstricci\u00f3n \/ dilataci\u00f3n a lo largo de la arteriola.<\/p>\n<p>Demostramos que el ascorbato inyectado en ratones s\u00e9pticos previene la vasoconstricci\u00f3n conducida alterada al inhibir el NO derivado de la sintasa de \u00f3xido n\u00edtrico neuronal, conduciendo a un acoplamiento el\u00e9ctrico inter-endotelial restaurado a trav\u00e9s de uniones gap que contienen conexina 37. La hipoxia \/ reoxigenaci\u00f3n (factor de confusi\u00f3n en la sepsis) tambi\u00e9n altera el acoplamiento el\u00e9ctrico por la desfosforilaci\u00f3n de la conexina 40 dependiente de la prote\u00edna quinasa A (PKA); el ascorbato restaura la activaci\u00f3n de PKA necesaria para este acoplamiento.<\/p>\n<p>Ambos efectos del ascorbato podr\u00edan explicar su capacidad para proteger contra la hipotensi\u00f3n en la sepsis. El taponamiento capilar en la sepsis implica la adhesi\u00f3n plaquetaria-endotelial mediada por P-selectina y la formaci\u00f3n de microtrombos. La inyecci\u00f3n temprana de ascorbato previene el taponamiento capilar al inhibir la adhesi\u00f3n plaquetaria-endotelial y la expresi\u00f3n de P-selectina en la superficie endotelial.<\/p>\n<p>El ascorbato tambi\u00e9n previene la agregaci\u00f3n plaquetaria inducida por trombina y la expresi\u00f3n de P-selectina en la superficie plaquetaria, evitando as\u00ed la formaci\u00f3n de microtrombos. La inyecci\u00f3n retardada de ascorbato invierte el taponamiento capilar y la adhesi\u00f3n plaquetaria-endotelial; tambi\u00e9n aten\u00faa la ca\u00edda inducida por la sepsis en el recuento de plaquetas en la sangre sist\u00e9mica. La liberaci\u00f3n de plaquetas inducida por trombina del activador-inhibidor-1 del plasmin\u00f3geno (evento antifibrinol\u00edtico en la sepsis) es inhibida por el ascorbato de manera dependiente del pH.<\/p>\n<p>Por lo tanto, en condiciones acid\u00f3ticas en la sepsis, el ascorbato promueve la disoluci\u00f3n de los microtrombos en los capilares. Proponemos que la conducci\u00f3n arteriolar protegida \/ restaurada y la perfusi\u00f3n del lecho capilar por ascorbato contribuyen a reducir la lesi\u00f3n de \u00f3rganos y mejorar la supervivencia en la sepsis.<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Tyml, K. (2017).\u00a0Vitamin C and microvascular dysfunction in systemic inflammation.\u00a0Antioxidants, 6(3), 49. La sepsis, disfunci\u00f3n org\u00e1nica potencialmente mortal causada por una respuesta disfuncional del hu\u00e9sped a la infecci\u00f3n, se asocia con una alta mortalidad. Una estrategia prometedora para mejorar el resultado es inyectar a los pacientes por v\u00eda intravenosa con ascorbato (vitamina C). En modelos [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[3],"tags":[],"class_list":["post-44217","post","type-post","status-publish","format-standard","hentry","category-naturopatia-alimentaria-trofologia"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v21.8 - https:\/\/yoast.com\/wordpress\/plugins\/seo\/ -->\n<title>\u00c1rea de conocimiento: Tecnolog\u00eda Naturop\u00e1tica o Naturopat\u00eda Aplicada: M\u00f3dulo: Naturopat\u00eda Alimentaria - TROFOLOGIA: &quot;Vitamina C y disfunci\u00f3n microvascular en la inflamaci\u00f3n sist\u00e9mica&quot; -<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/rina.naturopatiadigital.eu\/?p=44217\" \/>\n<meta property=\"og:locale\" content=\"es_ES\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"\u00c1rea de conocimiento: Tecnolog\u00eda Naturop\u00e1tica o Naturopat\u00eda Aplicada: M\u00f3dulo: Naturopat\u00eda Alimentaria - TROFOLOGIA: &quot;Vitamina C y disfunci\u00f3n microvascular en la inflamaci\u00f3n sist\u00e9mica&quot; -\" \/>\n<meta property=\"og:description\" content=\"Tyml, K. (2017).\u00a0Vitamin C and microvascular dysfunction in systemic inflammation.\u00a0Antioxidants, 6(3), 49. La sepsis, disfunci\u00f3n org\u00e1nica potencialmente mortal causada por una respuesta disfuncional del hu\u00e9sped a la infecci\u00f3n, se asocia con una alta mortalidad. Una estrategia prometedora para mejorar el resultado es inyectar a los pacientes por v\u00eda intravenosa con ascorbato (vitamina C). 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(2017).\u00a0Vitamin C and microvascular dysfunction in systemic inflammation.\u00a0Antioxidants, 6(3), 49. La sepsis, disfunci\u00f3n org\u00e1nica potencialmente mortal causada por una respuesta disfuncional del hu\u00e9sped a la infecci\u00f3n, se asocia con una alta mortalidad. Una estrategia prometedora para mejorar el resultado es inyectar a los pacientes por v\u00eda intravenosa con ascorbato (vitamina C). 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