{"id":45242,"date":"2020-12-19T06:30:06","date_gmt":"2020-12-19T05:30:06","guid":{"rendered":"https:\/\/rina.naturopatiadigital.eu\/?p=45242"},"modified":"2020-12-19T22:57:27","modified_gmt":"2020-12-19T21:57:27","slug":"45242","status":"publish","type":"post","link":"https:\/\/rina.naturopatiadigital.eu\/?p=45242","title":{"rendered":"\u00c1rea de conocimiento: Tecnolog\u00eda Naturop\u00e1tica o Naturopat\u00eda Aplicada: M\u00f3dulo: Naturopat\u00eda Alimentaria &#8211; TROFOLOGIA: \u00abEl \u03b1-tocoferol inhibe la agregaci\u00f3n de plaquetas humanas mediante un mecanismo dependiente de la prote\u00edna quinasa C\u00bb"},"content":{"rendered":"<p><a href=\"https:\/\/1.bp.blogspot.com\/-syn9KWtFtgs\/X952nW_x_iI\/AAAAAAAAhRo\/IoEh2jz1saIkEeWPC_WvYcCOrIEWeWNyQCLcBGAsYHQ\/s200\/Circulation.png\"><img decoding=\"async\" class=\"alignleft\" src=\"https:\/\/1.bp.blogspot.com\/-syn9KWtFtgs\/X952nW_x_iI\/AAAAAAAAhRo\/IoEh2jz1saIkEeWPC_WvYcCOrIEWeWNyQCLcBGAsYHQ\/s0\/Circulation.png\" border=\"0\" data-original-height=\"200\" data-original-width=\"200\" \/><\/a>Freedman, J. E., Farhat, J. H., Loscalzo, J., &amp; Keaney, J. F. (1996). <a href=\"https:\/\/www.ahajournals.org\/doi\/full\/10.1161\/01.CIR.94.10.2434\">\u03b1-Tocopherol inhibits aggregation of human platelets by a protein kinase C\u2013dependent mechanism<\/a>.\u00a0<i>Circulation<\/i>, 94(10), 2434-2440.<\/p>\n<p>ANTECEDENTES: Los estudios epidemiol\u00f3gicos indican que la vitamina E (\u03b1-tocoferol) ejerce un efecto beneficioso sobre las enfermedades cardiovasculares. El efecto de la vitamina E se ha atribuido generalmente a su actividad antioxidante y a la protecci\u00f3n antioxidante de las LDL. A diferencia de su efecto sobre las LDL, tambi\u00e9n se sabe que la vitamina E inhibe la agregaci\u00f3n y la adhesi\u00f3n plaquetarias in vitro, pero se desconocen los mecanismos responsables de estas observaciones.<\/p>\n<p>METODOS Y RESULTADOS: Utilizando plaquetas filtradas en gel derivadas de plasma rico en plaquetas tratado con \u03b1-tocoferol (500 \u03bcmol \/ L) o veh\u00edculo (etanol al 0,5%), encontramos que la inhibici\u00f3n de la agregaci\u00f3n plaquetaria por \u03b1-tocoferol estaba estrechamente relacionada con su incorporaci\u00f3n. en plaquetas (r = -. 78; P &lt;.02). La incorporaci\u00f3n plaquetaria de \u03b1-tocoferol se asoci\u00f3 con una reducci\u00f3n significativa de la sensibilidad plaquetaria a la agregaci\u00f3n por adenosina 5&#8242;-difosfato, \u00e1cido araquid\u00f3nico y \u00e9ster de forbol (PMA) en aproximadamente 0,15, 2 y 100 veces, respectivamente. Por el contrario, las plaquetas tratadas de forma similar con hidroxitolueno butilado, otro potente antioxidante soluble en l\u00edpidos, no demostraron ning\u00fan cambio en la sensibilidad a estos agentes. La incorporaci\u00f3n de plaquetas de \u03b1-tocoferol inhibi\u00f3 la estimulaci\u00f3n inducida por PMA de la prote\u00edna quinasa C de plaquetas (PKC), determinada por fosforilaci\u00f3n del sustrato de PKC de 47 kD. En 15 sujetos normales, la suplementaci\u00f3n oral con \u03b1-tocoferol (400 a 1200 UI \/ d) result\u00f3 en un aumento en el contenido de \u03b1-tocoferol plaquetario que se correlacion\u00f3 con una marcada inhibici\u00f3n de la agregaci\u00f3n plaquetaria mediada por PMA (r = .67; P &lt;.01 ). Las plaquetas derivadas de estos sujetos despu\u00e9s de la suplementaci\u00f3n tambi\u00e9n demostraron una aparente inhibici\u00f3n completa de la estimulaci\u00f3n de PKC por PMA.<\/p>\n<p>CONCLUSIONES: Estos datos indican que la incorporaci\u00f3n plaquetaria de \u03b1-tocoferol en los niveles alcanzados con la suplementaci\u00f3n oral est\u00e1 asociada con la inhibici\u00f3n de la agregaci\u00f3n plaquetaria a trav\u00e9s de un mecanismo dependiente de PKC. Estas observaciones pueden representar un mecanismo potencial para el efecto beneficioso observado del \u03b1-tocoferol en la prevenci\u00f3n del desarrollo de la enfermedad de las arterias coronarias.<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Freedman, J. E., Farhat, J. H., Loscalzo, J., &amp; Keaney, J. F. (1996). \u03b1-Tocopherol inhibits aggregation of human platelets by a protein kinase C\u2013dependent mechanism.\u00a0Circulation, 94(10), 2434-2440. ANTECEDENTES: Los estudios epidemiol\u00f3gicos indican que la vitamina E (\u03b1-tocoferol) ejerce un efecto beneficioso sobre las enfermedades cardiovasculares. El efecto de la vitamina E se ha atribuido generalmente [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[3],"tags":[],"class_list":["post-45242","post","type-post","status-publish","format-standard","hentry","category-naturopatia-alimentaria-trofologia"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v21.8 - https:\/\/yoast.com\/wordpress\/plugins\/seo\/ -->\n<title>\u00c1rea de conocimiento: Tecnolog\u00eda Naturop\u00e1tica o Naturopat\u00eda Aplicada: M\u00f3dulo: Naturopat\u00eda Alimentaria - TROFOLOGIA: &quot;El \u03b1-tocoferol inhibe la agregaci\u00f3n de plaquetas humanas mediante un mecanismo dependiente de la prote\u00edna quinasa C&quot; -<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/rina.naturopatiadigital.eu\/?p=45242\" \/>\n<meta property=\"og:locale\" content=\"es_ES\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"\u00c1rea de conocimiento: Tecnolog\u00eda Naturop\u00e1tica o Naturopat\u00eda Aplicada: M\u00f3dulo: Naturopat\u00eda Alimentaria - TROFOLOGIA: &quot;El \u03b1-tocoferol inhibe la agregaci\u00f3n de plaquetas humanas mediante un mecanismo dependiente de la prote\u00edna quinasa C&quot; -\" \/>\n<meta property=\"og:description\" content=\"Freedman, J. E., Farhat, J. H., Loscalzo, J., &amp; Keaney, J. F. (1996). \u03b1-Tocopherol inhibits aggregation of human platelets by a protein kinase C\u2013dependent mechanism.\u00a0Circulation, 94(10), 2434-2440. ANTECEDENTES: Los estudios epidemiol\u00f3gicos indican que la vitamina E (\u03b1-tocoferol) ejerce un efecto beneficioso sobre las enfermedades cardiovasculares. 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